Romanesque Cauliflower- Speshul Ted
Romanesque Cauliflower- Speshul Ted

The Concept of Gender Specific Meals

      Many foods naturally produce anti-masculine chemicals. Eating these foods will result in a lowered male hormone level and sexual function. These same foods will benefit females. Separating meals would allow men and women to develop their hormones in the direction they are meant to. This concept already happens to a degree with stereotypes and also commonly happened in paleolithic times.
     Restaurants and food packaging companies need to use this idea and work with ingredients of their specific purpose. Without this concept, people will continue to be affected negatively by the food they eat.

“Major Hormones:
DHT: A Derivative of Testosterone responsible for Masculinity
IGF-1: Main hormone responsible for Height
Estrogen: Main hormone responsible for Femininity”

Sous Vide New York Strip Steak- Austin Matherne
Sous Vide New York Strip Steak- Austin Matherne

Masculine Foods

     Different types of food directly alter daily hormone levels.  Much of the American diet includes fried oils which lower DHT. Cooking oils are often the main source of feminizing foods. Most plants contain compounds which decrease male hormones as the plants primary self defense.

List of Fats in Descending Benefit

Beef Fat

     The fat from a cow contains oleyl alcohol. This compound actually increases DHT production. In addition to this compound, beef fat contains the perfect types of fatty acids for natural DHT production.

Modern Butter

     Butter contains the right types of fatty acids to produce natural DHT levels. In contrast, Grass Finished butter actually causes a decrease in DHT production due to its higher omega 3 fatty acid content. The best type would be from cows with no added hormones because often they use estrogen to fatten up the cow and this carries into the butter.

Extra Virgin Olive Oil

     Olive Oil is the least safe fat source for men. Olive Oil does in fact lower DHT formation due to its high Oleic acid content. It is unique in that it counter acts this inhibition. A chemical in olive oil called Oleuropein increases testosterone as well as lowers estrogens creation.


List of Foods in Descending Benefit

Smoked Beef

     Smoked beef is the perfect food for DHT.  Beef contains creatine which has been shown to increase DHT levels. Wood Smoked meats also contain a chemical called alpha-Naphthoflavone which has been found to decrease estrogen and indirectly increase DHT.


    This grain is eaten as food as well popped in a similar way as popcorn. Sorghum has been found to increase DHT levels directly.

Portobello Mushrooms

    This special type of mushroom is perfect for DHT. Eating portobello mushrooms has been shown to decrease estrogen and indirectly increase DHT. Portobello mushrooms should be well cooked to remove a mild toxin present in them.
Grilled Portobello Mushrooms- Joshua Bousel
Grilled Portobello Mushrooms- Joshua Bousel


    This stalk is very good for DHT production. It is the highest source of a hormone called androsterone. This is a DHT precursor and will lead to increased DHT levels.

Chicken Heart

    This food is a perfect food for DHT. Chicken heart contains one of the highest sources of phosphatidylserine. This chemical has been found increase DHT production in the body.


    This starch is perfect to maintain DHT levels. It doesn’t impact DHT levels to the extent that other carbohydrate sources do. When made into fries, the best oil would be olive oil.


    Chicken eggs are great for DHT. They contain cholesterol which is a building block of DHT. Non Grass fed chickens are better because they contain less DHA which is a DHT inhibitor.

Rye Bread

    The fatty acids contained in rye are relatively good as a carbohydrate source. The bread should be made with butter or meat fat as most breads are made with soybean oil.
Rye Bread-Rebecca Winzenried

Cows Milk

    Milk is a great beverage for IGF-1. Cows milk stimulates the release of IGF-1. The saying milk is good for your bones is true due to its high calcium content as well as its ability to increase IGF-1.

Green Beans

    Beans are a decent food source for DHT. They are a rich source of Zinc which is a testosterone precursor. Zinc actually inhibits DHT production but also inhibits DHT destruction. This makes it overall pretty good for DHT levels.

List of Drinks in Descending Benefit

Whiskey Barrels- ScottSimPhotography
Whiskey Barrels- ScottSimPhotography


     Dark Coffee has been shown to increase DHT levels. Caffeine in coffee causes the body to increase all hormones in long term use. Sorghum was used as a coffee alternative and pairs great with coffee as both increase DHT levels.


     This liquor is good for DHT. Many recipes contain sorghum which has been found to increase DHT.


List of Activities in Descending Benefit

Tobacco Use

    Nicotine is the strongest DHT booster. Nicotine inhibits DHT’s breakdown. Tobacco raises DHT best when used over long periods of time and in high amounts. This is because half of the nicotine in your body is broken down into cotinine every two hours. Cotinine also inhibits DHT’s breakdown and accumulates in the body over long term use. Half of the cotinine in your body is excreted every 20 hours. This combined effect effectively increases DHT in the body.


    Chronic exercise has been found to increase all hormones. Weight lifting increases DHT and testosterone, while running increases growth hormone and decreases estrogen.
Splash!- Luci Correia
Splash!- Luci Correia

Feminine Foods

   The majority of foods that are available are good for females. They lower male DHT and increase female estrogen.

List of Fats in Descending Benefit

Safflower Oil

   Safflower oil is best oil for women because it decreases DHT. It has a high oliec acid content as well as a high level of betasitosterol. Both of which will lower DHT.

Coconut Oil

   Oil derived from coconuts are beneficial for women due to its high lauric acid content. Coconut oil will lower DHT levels in the body. This will indirectly increase estrogen levels.

Fish Oil

   Fish Oil lowers DHT in the body. DHA is responsible for blocking DHT production. Krill oil also inhibits DHT to a greater extent because it contains Zeaxanthin.

Soybean Oil

   Soybean Oil has a high oliec acid content as well as a high linoleic acid which both lower DHT and lead to increased estrogen..

Canola Oil

   Canola Oil contains decent levels of oliec acid as well as linoleic acid. Both of these will lower DHT and lead to increased estrogen.
Dark Chocolate Bar- Boz Bros
Dark Chocolate Bar- Boz Bros

List of Foods in Descending Benefit

Soy foods

    Almost any chinese prepared food contain soybeans. Soybeans have been found to increase estrogen and decrease DHT. This is perfect for females.

Reishi Mushrooms

    These chinese mushrooms act like estrogen when eaten. This is perfect for females.

Indian foods

    Curry spiced foods contain Turmeric. Turmeric has been found to lower DHT. Indian foods thus great for women.
Turmeric root used in indian foods- Steenbergs
Turmeric root used in indian foods- Steenbergs


    This algae has been seen as a superfood. It is great for women due to its high GLA content. GLA is one of the strongest DHT inhibitors.

Chia Seeds

    Chia seeds are found in certain drinks like kombucha and foods like salads and bread. They lower DHT because of their high ALA content.

Flax Seeds

    Much like Chia seeds, Flax seeds lower DHT due to their high ALA content and are used in similar foods.
Fresh Tomatoes -Adam Selwood
Fresh Tomatoes –Adam Selwood


   Fresh tomatoes lower DHT due to a chemical called lycopene. Lycopene also decreases IGF-1’s destruction. This makes tomatoes decent for females. Cooking tomatoes lowers the lycopene content quite a bit. Organic Tomatoes also contain less lycopene than non-organic ones.


     Cocoa contains EGCG which lowers estrogen, Testosterone, DHT, and IGF-1. Cocoa also contains theobromine which is known to kill testes cells. Chocolates main effects are on decreasing DHT and thus is good for females.


     Chicken breast contains a relatively decent amount of Oleic acid which lowers DHT.

Broccoli, Brussels Sprouts, Kale, and Cauliflower

   These plants are the least safe for women. These plants are known to inhibit estrogen production due to indole-3-carbinol. It counteracts this by also inhibiting testosterone function. The high zinc content also decreases DHT’s destruction.

List of Drinks in Descending Benefit


    This spirit is the perfect beverage for women. Gin contains Juniper Berries and Star Anise. Both of which have strong acting estrogen compounds. Alcohol has also been found to increase estrogen response.


    Red wine has been shown to increase estrogen in women. Alcohol also has been shown to increase estrogen response.

Green Tea

    Green tea contains EGCG which lowers estrogen, Testosterone, DHT, and IGF-1. This is decent for women because it mainly lowers DHT.

List of Activities in Descending Benefit

Birth Control

    The pill is the strongest source of estrogen because it is pure synthetic estrogen. This is the holy grail for women because it increases what women are meant to have. [Dr. Cremers’ comment: I would not advocate anyone taking external hormones unless they have had a full eye exam given risk of vein or artery occlusion [and not just of the eye] which I have seen many of in my career). 

Tea Tree Oil Shampoo

    Many shampoo’s and conditioners contain tea tree oil. This oil increases estrogen and is absorbed through contact with skin. Nearly all other female shampoos also contain estrogenic or anti-DHT components.


   Perfume is a staple for women. Putting it on leads to inhalation and absorption of estrogenic chemicals called phtalates. Phtalates also decrease testosterone response. Nearly all perfumes contain phtalates to maintain fragrance for longer.
Popular Perfume-Lindsay Wilson
Popular Perfume-Lindsay Wilson

Make up

    Make up also increases estrogen due to phthalates present in them.

Nail Polish

   Estrogenic Phthalates are also present nail polish which could be absorbed through inhalation or spills on skin during drying.


   Spandex could potentially increase estrogen if in the right circumstances. Tetrhydrofuran, the plastic-rubber in spandex, can be absorbed into the skin when dissolved into a common lotion containing cetostearyl alcohol. This would increase estrogen response in the body. This is perhaps the reason only girls wear spandex clothing.
A crop Field -Wazimu0
A crop Field -Wazimu0


    Most plants contain chemicals which affect hormones as a defense against predators. The effects of this exposure leads to less attractive characteristics. Even behavior is altered with exposure leading to less confidence and more anxiety. This is due to DHT’s anti anxiety effect that is similar to alcohol.
    Many different cultures have relied on local food sources to live. Chinese and Indian cultures have been living off of female specific foods and this has resulted in their apparent appearance and behavior. Asian’s have been found to have a lower risk of prostate cancer due to this.
    We already see social groups sticking to certain gender safe diets. Southern Culture is known for steak, potatoes, green beans, and portobello mushrooms. All of which adhere to masculinity specific foods.

Separate Entrées

    Restaurants receive stereotypes from society due to the feedback their food has given to their customers. Starbucks is seen as a female business probably due to their high use of chocolate in their products. Sushi Restaurants are also seen as female restaurants due to the high soy content in the food. Panera is often seen as a female restaurant because of its heavy use of estrogenic foods.
    The future of food involves gender separation of meals. Food packaging companies need to use ingredients that are separated by gender. This will lead to restaurants implementing this concept. With Gender specific meals, foods don’t need to be changed, just some of the ingredients involved.
   Without separate meals, society will continue to be influenced in negative ways. Homosexuality could easily beinfluenced by a strict opposite gender diet. In fact there is evidence that over generations, this effect would become more obvious.
   Epigenetic studies have shown that drugs and foods can influence the hormones of a family line. Having high androgen levels in the first few days of fertilization can lead to similar high genetic androgen response. This effect carries on through at least 2 generations and some studies show it be true up to 4 generations. This same idea can work with low androgen levels and lead to a feminine identity.
Food processor- Steve Snodgrass
Food processor- Steve Snodgrass


USDA Food Composition Search

Anti-androgenic activity of fatty acids.

“5a-Reductase Inhibitory Activity. Fig. 1 shows that only certain saturated fatty acids
have inhibitory activity. Among the saturated fatty acids, the inhibitory acids (more
than 50% inhibition at 1.3 mm) have C
12–C16 chains. The relative inhibitory potencies
for the 5a-reductase of saturated fatty acids are, in decreasing order, C12> C14>C15>C16> C18. This result suggested that the 5a-reductase inhibitory activity of saturated
fatty acids depends on the hydrocarbon-chain length. ”


“Anti-DHT Fatty Acid Strength in Decreasing order:

gamma-linolenic acid

DHA = cis-6,9,12,15-octatetraenoic acid = arachidonic acid = alpha-linolenic acid

linoleic acid

palmitoleic acid

oleic acid

myristoleic acid”


Cows Milk

“The IGF-1 may be either absorbed from milk, or stimulated by its ingestion, or both.”


Smoked Beef

“ AhR is involved in xenobiotic metabolism and in mediating the toxic effects of dioxin-like compounds. Crosstalk has been observed among AhR and nuclear receptors, but has been most well studied with respect to ER signaling. Activated AhR inhibits ER activity through a number of different mechanisms, whereas ERα has been reported to have a positive role in AhR signaling. ”


Chicken Heart

“Among these endogenous phospholipids, only phosphatidylserine stimulated the 5 alpha-reductase, suggesting that the lipid requirement is specific for phosphatidylserine in steroid 5 alpha-reductase from liver microsomes.”



“ Our results show that caffeine intake increased the concentrations of T and DHT, organ weight, epithelial cell proliferation and AR tissue expression in the ventral prostatic lobe. ”


Epigenetic Studies

“Androgen excess is a central feature of polycystic ovary syndrome (PCOS), which affects 6% to 10% of young women. Mammals exposed to elevated androgens in utero develop PCOS-like phenotypes in adulthood, suggesting fetal origins of PCOS.”


“the plastic-derived endocrine disruptor compounds bisphenol-A (BPA), bis(2-ethylhexyl)phthalate (DEHP), and dibutyl phthalate (DBP) triggered pubertal abnormalities, testis disease, obesity, and ovarian disease (primary ovarian insufficiency and polycystic ovaries) in animals of the F3 generation, while kidney and prostate disease were observed only in the F1 generation”


“There were striking sex-specific transmissions, such that the food supply of the paternal grandfather was associated with the mortality rate of grandsons only, while the early-life food supply of the paternal grandmother was only associated with the mortality rate of granddaughters 2, 3 and 4.

Offspring of maternal adverse experience (such as violence, stress, poverty) which triggers a pattern of behavior in the offspring (e.g., aggression) would create the same adverse experience (aggression) for their offspring, leading to perpetuation of the phenotype across generations Thus, epigenetic processes might be involved not only in creating differences between tissues but also among individuals.”

Thumbnail Source –Michael Bentley
5. Good blog:

4 Causes of Androgen Excess in Women

Why some women have too much testosterone.If you suffer hair loss, facial hair (hirsutism), oracne, then you know all about androgen excess. Androgen means “male-hormone”, and your doctor may have gone so far as to test you for testosterone and other androgens. Or she may not have bothered to test, and simply diagnosed you based on symptoms. Either way, she probably prescribed an androgen-suppressing drug such as spironolactone.
But let’s think this through a bit more. Why do you have androgen excess? What else can you do about it?

Androgenic Birth Control

The simplest and most preventable cause of androgen excess is hormonal birth control. Some (not all) types of birth control use synthetic progestins that have a “high androgen index”, which means they are testosterone-like. They are androgenic and promote hair-loss, and this effect is well known amongst the medical community although rarely mentioned to patients.
Are you taking “testosterone-like” birth control? Read the label.
Progestins with a high androgen index include medroxyprogesterone (Depo-provera injection), levonorgestrel (many pills, Norplant implant, Mirena IUD, and morning-after pill), norgestrel, and etonogestrel (many pills, Nuvaring, Nexplanon implant).
Progestins with a low androgen index include drospirenone (which is spironolactone),norgestimate, and cyproterone, and of course natural progesterone. In fact, natural, endogenous progesterone is anti-androgenic because inhibits 5 alpha-reductase ,which is why natural progesterone is good for hair.
Treatment is to switch birth control. Choose a less androgenic progestin, or even better: Choose a non-hormonal method such as Fertility Awareness Method, condoms, or copper IUD. Once you stop the progestin, your androgen symptoms should subside, but unfortunately, hair loss can sometimes endure for months or even years.

Hypersensitivity to a Normal Amount of Androgen

A slightly more perplexing cause of excess androgens is an apparent hypersensitivity of the androgen receptor. This is the diagnosis when there are high androgen symptoms butnormal levels of androgens on a blood test. When hair loss is the main symptom, androgen hypersensitivity is called androgenic (or androgenetic) alopecia.
Androgen hypersensitivity is surmised to be genetic, but that explanation is not very satisfactory because previous generations of young women did not suffer the same epidemic of hair loss. A more likely explanation is either exposure to androgenic progestins (discussed above), or intriguingly: Inflammation at the androgen receptor, as explored in this 2011 research.  I have found that my patients’ androgen-sensitivity improves when they reduce chronic inflammation.
Treatment is to reduce sensitivity of the androgen receptor by reducing inflammation. You can do that by: 1) Not smoking, 2) Avoiding inflammatory foods such as sugar, wheat, anddairy products, 3) Eating anti-inflammatory vegetables (phytonutrients have been shown to improve female hair loss), 4) Reestablishing healthy intestinal bacteria, and 5) Supplementing with the anti-inflammatory mineral zinc.

Adrenal Androgen Excess

Your adrenal glands make about 50 percent of your total androgens. You can estimate how much androgen is coming from your adrenal glands by measuring a hormone called dehydroepiandrosterone sulfate (DHEA-S) on blood test. If only DHEA-S is elevated, but not testosterone or androstenedione, then you have predominantly adrenal androgen excess. If you have elevated DHEA-S, testosterone, and androstenedione, then you fall into the category of ovarian androgen excess discussed below.
One cause of adrenal androgen excess is a relatively common genetic condition called non-classic (or late-onset) congenital adrenal hyperplasia (NCAH), which is diagnosed by a blood test for a hormone called 17-hydroxyprogesterone.
Treatment of congenital adrenal hyperplasia is low dose hydrocortisone to down-regulate adrenal androgen production. Interestingly, low dose hydrocortisone was also historically used to treat PCOS, and is still used by some doctors today.
Adrenal androgen excess is also the key feature in about 20 percent of women diagnosed with polycystic ovarian syndrome (PCOS). Predominantly adrenal-androgen PCOS is quite a different condition from ovarian-androgen PCOS (see below), in that it is not driven by insulin resistance or underlying conditions that impair ovulation. It is driven by factors that affect the adrenal glands, such as stress. I truly hope that PCOS’s upcoming name changewill distinguish between adrenal-androgen PCOS and ovarian-androgen PCOS, and therefore save us all a lot of confusion.
Proposed causes of adrenal-androgen PCOS include genetics, and stress around the time of puberty. I strongly suspect that endocrine disrupting chemicals (EDCs) also play a role, but that research is still in its infancy.
Treatment of adrenal-androgen PCOS is to normalise adrenal function. This is probably the type of PCOS that responded to the low-dose cortisone historically prescribed for PCOS. Adrenal-androgen PCOS also responds to stress-reduction, and to hypothalamic-pituitary-adrenal (HPA) regulating supplements such as magnesium, phosphatidylserine, and rhodiola. Adrenal-androgen PCOS may also require androgen blocking supplements such as diindolylmethane (DIM). For a full discussion of adrenal-androgen PCOS, please see Fiona McCulloch’s post: Treating adrenal androgen excess.

Ovarian Androgen Excess

Now we come to a condition that is synonymous with androgen excess in women: Polycystic ovarian syndrome (PCOS). The key defining feature of PCOS is ovulatory dysfunction, and the over-production of testosterone and androstenedione by the ovaries.
PCOS is not an ovarian disease per se. The ovulatory dysfunction is an expression or symptom of a larger set of underlying hormonal and metabolic problems such as insulin resistance and inflammation.
But don’t ovarian cysts cause PCOS?
Polycystic ovarian syndrome got its name from the way ovaries look on ultrasound. You would be forgiven then for thinking the small multiple follicles cause the condition, but they don’t. They are simply an indicator that ovulation did not occur that month. Any woman can have polycystic ovaries on occasion, including a woman on the pill, and a woman with perfectly normal hormones. Don’t be misdiagnosed by an ultrasound. See the Surprising Truth About PCOS.
Is PCOS genetic?
Yes, researchers suspect that PCOS is influenced by genes, and possibly also by exposure to androgens or environmental toxins in utero. That is a depressing thought, because it might mean you were born with the condition. It doesn’t work like that. A genetic tendency does not mean you will always suffer the symptoms. You can modify your genetic expression with diet, lifestyle, and other natural treatments.
Treatment of ovarian-androgen PCOS is to correct the underlying insulin resistance (or other metabolic problem) and thereby reestablish regular ovulation. That usually means quitting sugar, and using insulin-sensitising supplements such as myo-inositol, magnesium, and berberine. Exercise and the diabetic drug metformin are also helpful. Non-insulin-resistant types of PCOS can benefit from the androgen-reducing herbal formula Peony & Licorice.  See my book Period Repair Manual and 4 Types of PCOS.
What is your experience with androgen excess? What has helped you?
Yours in Health,
Another interesting post:

How DHT and Testosterone Affect Your Acne

Do DHT and testosterone cause acne?If you’ve been researching acne then you’ll almost certainly have come across testosterone and DHT.
These androgens are touted as being the biggest villains behind acne ever. It does seem logical – after all, these hormones increase massively during puberty, and the first pimples appear in puberty too.
Hence, many acne patients are attempting to reduce their DHT and testosterone levels whether through drugs or other therapies. Many foods like green tea, peppermint tea, and saw palmetto are recommended by acne gurus for crushing androgen levels.
Perhaps you too have been recommended them.However, reducing DHT and testosterone for acne is a strategy I cannot endorse, at least not for men, mainly because they are important for many aspects of health.
The full story on these acne hormones is more complicated – they are far more benign that their reputation suggests. DHT and testosterone can cause acne, but they far from guarantee it.
How DHT/testosterone cause acne
Testosterone and DHT are the two strongest androgens in the human body. They are behind many of the classic effects that kick in during puberty; growing muscle mass, rabid sex drive, new body hair, a deeper voice, and all the rest.
Both testosterone and DHT activate these characteristics by stimulating your androgen receptors. A DHT molecule, for example, causes body hair growth by binding to receptors in the hair follicles, which triggers the hair to grow or grow faster if it’s already growing. A large proportion of the startling changes we undergo in our teenage years occur thanks to these hormones…
…and one of the biggest changes is spotty skin full of acne. DHT and testosterone kick off the whole acne saga in the first place, by making your skin oily. Androgens bind to the receptors in your sebaceous glands, which has a huge stimulating effect; the glands dramatically rev up their output of sebum.
You’ll know the horror story that follows; your pores get blocked, p.acnes bacteria shows up, and then it’s only a matter of time before acne rears its head. That’s also why annoyed teenagers develop oily skin at about age 13. DHT/testosterone have few other acne causing powers – increased sebum production is nearly the only factor.
Nevertheless, it’s extremely significant because without blocked skin pores, it’s a lot more challenging for p.acnes bacteria to multiply. Hence, there’s no inflammatory response to them, and acne will struggle to be born at all.
However it’s not a simple, linear case of “more androgens = more acne”. DHT is a substantially stronger hormone than testosterone, especially for your skin.  
DHT – also known as dihydrotestosterone, this is the most powerful androgen in the human body by some distance. When one thinks of juiced-up bodybuilders and their sex-crazed personalities, steroids or testosterone are the first words that spring to mind.
However it is actually DHT that is responsible for the vast majority of male sex characteristics.  The androgen receptors for body hair, sex drive, and importantly for us, the sebaceous glands, all lie under DHT’s jurisdiction. It is DHT that causes your skin to become oily and that’s why it’s most important for acne. However, testosterone still has a role in acne to play…
Testosterone – the king of androgens and the hormone that allows DHT to be born. Both men and women produce testosterone but men have far higher levels. Until a boy is roughly 9 years old, his testosterone will be in the range of 7-20 ng/DL. By the end of puberty though, these levels will have skyrocketed to between 270-1070.
Hence, he grows into a man with dramatically increased muscle mass, changed facial bone structure, and all the other changes that rapidly occur in as little as 5 years.
However, what’s interesting about testosterone is that the only effect it produces directly is increased muscle mass. That’s why bodybuilders use steroids; they are basically synthetic forms of testosterone, and are optimal for getting gigantic muscles. Every single other effect that’s associated with testosterone is actually due to more DHT being produced.
All DHT is converted from testosterone, by an enzyme called 5-alpha reductase. The reason why higher testosterone gives you a deeper voice and increased body hair is not directly due to the testosterone itself but rather that because your testosterone is high, more will inevitably get converted to DHT. For acne, this means that having higher testosterone will still result in oilier skin, even if it doesn’t directly cause it.

The implications for acne
DHT, testosterone, androgens and acne. What this all means is that if you desire to reduce your sebum production by focussing on androgens, then DHT should be your main focus. Specifically you should focus on 5-alpha enzyme. It’s this enzyme that is responsible for the whole conversion process; without 5-alpha reductase there would be no DHT at all.
Inhibiting this enzyme is what most DHT reduction programs for acne focus on, including the ones favoured by acne patients. Green tea has a specific antioxidant called epigallocatechin-3-gallate that directly weakens the potency of 5-alpha reductase, and hence you’ll see many acne gurus recommend it. One supplement I recommend for lowering DHT (in certain circumstances) is saw palmetto.
If you were to go down this road then you wouldn’t have less overall androgens in your body; testosterone would actually increase as less is being used up in conversion. However DHT would decrease and that’s what matters. Your sebaceous glands would be less stimulated and eventually your pores would unblock, and finally your acne would improve significantly.
With that said, the DHT question is not that simple at all. For over half of acne patients, reducing DHT is totally unnecessary. In fact, doing so could wreck your health.
Whether you’re a man or a woman is absolutely vital for your approach to DHT and testosterone, because a man’s health depends on them to a far great extent. As discussed earlier, DHT (and testosterone) are needed for all the classic androgenic effects. Without adequate DHT levels you would have no sex drive, you would have no energy, your muscles would be weak and useless, and you would develop mental problems such as brain fog and maybe even depression.
In short, your quality of life would go massively downhill. For example:
  • In this 2014 study, scientists in Amirkola, Iran, took 830 thirty elderly men with a mean age of 70.02. 237 had some symptoms of depression, while 593 had no symptoms at all. Blood samples were collected and testosterone levels were analysed. The mean serum testosterone level in the men without depression was significantly higher. The men who were depressed were far more likely to have low testosterone levels. The scientists concluded that “the results of this study showed a significant inverse relationship between serum testosterone levels and depressive symptoms in elderly men.
If you are a man who values the enjoyment of life even slightly, then I do not recommend any acne strategies for lowering DHT levels, lowering testosterone, or inhibiting 5-alpha reductase. You would improve acne, but you’ll have a whole new world of problems to tackle.
Moreover, you can still have high levels of DHT/testosterone and cure your acne. The acne problems can be neutralised easily.
The hormone insulin is just as potent at stimulating your sebum production; it also leads to the production of another hormone called IGF-1 that is again very potent. You can lower insulin levels simply by eating less carbohydrates, and that strategy won’t hurt you. In fact, lowering insulin will improve your health as high levels are a sign that you’re about to become diabetic.
Insulin also enhances the sensitivity of your skin’s androgen receptors. Therefore, by lowering your levels of insulin, androgens will be far less damaging for acne anyway. Additionally, there are numerous non-hormonal strategies; eating vitamin A rich-foods like sweet potatoes and eggs can reduce sebum production and acne significantly.
If you’re a woman, on the other hand, then androgens are far less important for your well-being. Women do need some testosterone/DHT since particularly low levels can cause depression and tiredness just like with men, but they need far less.
That’s why if you’re female, than reducing DHT is a viable option for acne, since you won’t destroy the rest of your health. Also, a woman’s acne is far more likely to be due to high DHT in the first place, because they are more sensitive to the effects of androgens.
There’s one story floating round the internet on men’s nutritional forums that illustrates this well. Supposedly there was a married man taking testosterone replacement therapy and he was applying testosterone cream to his skin. He was shocked to find that after touching his wife, within days she had grown facial hair. The amount of testosterone cream she absorbed from her husband’s skin would have been tiny, yet it was still enough to have masculinising effects.
This phenomenon means that the same amount of DHT can stimulate a lot more sebum production in women than men. Therefore while a man’s skin could withstand somewhat higher DHT levels, a woman’s would stand no chance. The female acne strategy should thus be to focus on reducing insulin as well as DHT levels, which will grant you a double whammy of sebum reducing power.
DHT/testosterone is not the biggest factor in acne!
DHT, antioxidants, testosterone and acne.Many members of the clear-skin community focus on androgens like they’re the single most important factor, but they’re not even close. Take the example of blocked pores. If your sebum production is high due to excessive DHT, then you might woefully believe that your pores are doomed to be blocked.,,
…however DHT does not have to be dangerous if you eat enough antioxidants. The biggest problem of oily skin is that it allows more of your sebum to oxidise, and eating plenty of anti-oxidant rich foods can prevent this. Your skin can easily cope with high androgens if you eat enough acne fruits, acne vegetables, herbs, spices, and dark chocolate.
Likewise, you can focus on insulin, and you can also focus on the other big cause of blocked pores: high keratin production. This protein glues dead skin cells together and helps them clog your pores. Vitamin A keeps keratin in check, as do other acne vitamins, and many environmental contaminants like arsenic can stimulate high production.
Consider your school days; you almost certainly had a teenage friend whose skin became very oily, yet still didn’t get acne. He or she probably had high DHT and testosterone, as most teenagers do, but unwittingly had a reasonably nutritious diet.
Maybe he ate plenty of meat, and thus lots of zinc, and that kept his inflammation down and meant that oily skin wasn’t a problem. Or maybe he played football a lot and was exposed to plenty of vitamin Dstimulating sunlight. The point to remember is this: the acne strategies for getting around high androgen levels are endless. In my 167-page eBook Annihilate Your Acne, we covered tons of factors such as sleep deprivation, stress levels, underused minerals like magnesium, and sun exposure.
You only have to look at history to assess that androgens don’t doom you to acne. Despite tons of acne patients worrying that their testosterone or DHT is too high, the modern man actually has less of both than ever before. Average testosterone levels have plummeted over the last century. There are some estimates that while the average testosterone level today ranges from 1070ng/DL to 270ng/DL, one hundred years ago the range was 2000-800!
Several studies have reported that testosterone levels have been dropping regardless of age; in other words a 60 year old man today has far lower levels than a sixty year old man 60 years ago.
If androgens were the biggest factor in acne then you would expect acne rates to have plummeted as well. However, by most accounts they have shot up; today it’s estimated that 40-55% of adults aged between 20 and 40 have acne. The adult acne epidemic is a very recent phenomenon.
This doesn’t mean that androgens are not a factor for acne. They are extremely important, but what this does tell us is that they do not override every healthy dietary and lifestyle choice you could make.
Conclusion – my final recommendations
If you are a woman, then placing plenty of effort into lowering androgens for acne is a very good idea.
If you have oily skin then you will particularly need to focus on reducing DHT via 5-alpha reductase inhibitors like green tea, or possible supplement with saw palmetto. You don’t want to go too low, because plenty of women do have testosterone deficiencies. Nevertheless, it’s a smart idea to drink an androgen blocker such as green tea or take this Saw Palmetto Berry Powder, which is the simplest and most effective DHT inhibitor I recommend on this website.
If you a man however, then I do not recommend these acne treatments at all, because DHT is far too important for the enjoyment of your life.
There’s a lot of men suffering from the effects of low testosterone or DHT these days. Fertility rates are plummeting, a lot of men are listless and can’t focus; you don’t want to join these guys. You might reduce a few pimples on your face, but if you don’t have the energy to enjoy the activities you love, then it’s pointless.
Remember that DHT doesn’t even have to be a problem. My skin is still oily and I hardly have any acne.Getting lots of nutrients, antioxidants, avoiding inflammatory foods, and reducing stress are far more important factors.
If you are a bodybuilder or any other athlete who needs high testosterone to be competitive, then acne free skin is well within your reach.