How to Loose Weight with Apple Cider Vinegar: Is Organic Apple Cider Vinegar better than Non-Organic

How to Best Loose Weight. 
         Does Apple Cider Vinegar Work: 
     Is Organic Apple Cider Vinegar better                         than Non-Organic


Many people are looking for the best way to lose weight safely. There are thousands of fads out there and many differing opinions–even from doctors. This makes advice hard to trust.

Apple Cider Vinegar is one of those rare ingredients which has almost no side effects (as long as you do not let is splash you in the eye or use on your skin: see last reference below), and has been shown to help control blood sugar and help in a solid weight loss plan.


Apple Cider Vinegar has many health benefits which have been published in journals (some are in the reference section).

Apple cider vinegar is widely used in salad dressings, marinades, vinaigrettes, food preservatives, chutneys, and other foods. 



The main classes of phenols in apples and Apple Cider Vinegar are:
a. flavonoids and 
b. polyphenolic compounds 

A number of studies have shown that these foods have a variety of pharmacological functions, including the below all of these potential positive benefits without adverse effects. 
1. Antioxidant activity (Yang et al. 2010; Denis et al. 2013): ACV increases antioxidants are essential to protect our cells from free radicals which can lead to mutations in DNA, early cell death, and cancer

2. Antidiabetic: Regulates Blood Sugar:(Shishehbor et al. 2008)
Also, Dr. Carol Johnston initially published a paper saying vinegar does not help improve blood sugar levels in 2009 (abstract is in References). But this professor and director of the Nutrition Department at Arizona State eventually presented a study at the 2013 Experimental Biology Conference in Boston saying: ingesting ACV at meal time increased satiety and reduced postprandial glycemia,

3. Cholesterol-lowering properties (Budak et al. 2011): 

Apple Cider Vinegar inhibits lipid peroxidation.
has a [rotective effect against erythrocyte (red blood cell), kidney, and liver oxidative injury, and lowered the serum lipid levels in mice fed high cholesterol, suggesting that it possesses oxidative stress scavenging effects, inhibits lipid peroxidation, and increases the levels of antioxidant enzymes and vitamins. (Nazıroğlu,et al. 2014)

4. Increases Antioxidant Enzyme Levels: (Nazıroğlu,et al. 2014)

5. Increases Vitamin Levels: (Nazıroğlu,et al. 2014) 

6. Protects against Erythrocyte (Red Blood Cell) Injury:

7. May decrease oxidative stress in body

8. Keep you pH Level Alkaline: 
Many chemical reactions and enzyme in your body work best at a particular pH. Some research has shown that high acid levels lead to a lack of energy and higher incidences of inflammation and infection. Eating too many carbohydrates and sugars makes your body more acidic and this promotes inflammation. Apple Cider Vinegar has been reported to help stabilize this. 


9. Lowers Blood Pressure and Possibly Heart Disease Risk:

Apple Cider Vinegar contains potassium, which is known to help balance the body’s sodium levels and maintain optimal blood pressure. It also contains magnesium, which helps to relax blood vessels walls, and ultimately lower blood pressure. Studies have shown it can increase HDL (good cholesterol) and decrease triglycerides, and prevent LDL (bad cholesterol) from being which leads to heart disease. oxidized which leads to heart disease.which leads to heart disease.

This is our favorite version below: Organic Apple Cider Vinegar








10. Helps with Detoxification of Liver and other Organs:

Studies show ACV helps flush out toxins that build up in the liver and possibly improve the natural blood filtration process of the blood. This can help boost energy levels. 

11. Improve Blood and Lymph Circulation:
ACV appears to detox the body by breaking up mucus and cleansing the lymph nodes, which allows for better lymph circulation. The lymphatic system removes toxins from cells and helps the immune system work properly.

12. Has Antifungal Properties and Stops Candida Overgrowth 

As ACV is rich in natural enzymes which can help eliminate candida in the body. Studies on dentures show ACV has anti-fungal properties (see below reference). Candida has been blamed for many different health issues, including poor memory, fatigue, headaches, depression, sugar cravings/addictions and yeast infections. While it’s a naturally occurring yeast, it can quickly grow out of control if the body becomes too acidic from eating too many carbs (like bread, cookies, cakes), processed foods and sugar, or if good bacteria is killed off from antibiotic use..

13. Works as a Probiotic for Gut Health & Can Help with Digestive Problems:

ACV stimulates digestive juices that help the body breakdown food. It can help indigestion, bloating, gas and heartburn. 1 teaspoon of ACV with a glass of water will bring relief from heartburn fast. Heartburn can occur in some patients from low stomach acid levels, and ACV helps bring that level up. Additionally, the healthy acids found in ACV, including acetic, isobutryic, lactic and propionic acid aids helps control the growth of unwanted yeast and bacteria in the stomach and throughout the body which can make one feel better. 
ACV does not help if you have a stomach ulcer which is usually due to a bacteria H. pylori and needs antibiotics. 

14. Increases Satiety & Accelerates Weight Loss

2 teaspoons of ACV to 16 ounces of water and sipping it throughout the day may help boost weight loss efforts. A paper published in Bioscience, Biotechnology, and Biochemistry showed that participants who consumed ACV for 12 weeks significantly decreased body weight, abdominal fat, waist circumference, and triglycerides. Further studies found drinking ACV every day for 12 weeks decreased participant’s visceral fat, lower waist circumference and BMI as compared to those who didn’t drink apple cider vinegar.
15. May Help to Prevent Osteoporosis
Osteoporosis, which means “porous bone,” is a bone disease due to many factors which cause a loss of bone density:  body loses too much bone, makes too little bone, or both. As a result, bones become weak and may break from a fall or, in serious cases, from sneezing or minor bumps.

One in 4 women in America over the age of 65 have been diagnosed with osteoporosis. Most patients have lost 50 to 75 percent of the original bone material from their skeleton when they are diagnosed. It affects 200 million women worldwide: about one of every 10 women aged 60, one of every five women aged 70, and two of every five women aged 80.

ACV aids in the nutrient absorption of calcium, an essential mineral for preventing osteoporosis, it’s one of the best tools you can use to reduce your risk. By drinking it along with each meal, it will help to ensure proper calcium absorption to strengthen your bones.

16. May Slow the Aging Process

The antioxidant properties in Apple Cider Vinegar and other vinegars can help prevent premature aging and even aid in slowing down the aging process. ACV’s ability to maintain the stability of the acid/alkaline balance in the body also helps.

17. Decreases Free Radical Damage

A Free Radical is any atom or molecule molecular species capable of independent existence with an unpaired electron in an atomic orbital. Many radicals are unstable and highly reactive and dangerous. They can either donate an electron to or accept an electron from other molecules, therefore behaving as oxidants or reductants. The most important oxygen-containing free radicals in many disease states are:
a. hydroxyl radical
b. superoxide anion radical
c. hydrogen peroxide
d. oxygen singlet
e. hypochlorite
f. nitric oxide radical
g. peroxynitrite radical. 

Free radicals are highly reactive species, capable, in the nucleus and in the membranes of cells, of damaging biologically relevant molecules such as DNA, proteins, carbohydrates, lipids.  Free radicals attack important macromolecules leading to cell damage/death and homeostatic disruption. Targets of free radicals include lipids, nucleic acids, and proteins but all cells are open game. 
It is thought that oxidative damage to cell membranes, tissues, proteins and even DNA, can result in diseases like ulcers, emphysema, arthritis and immune disorders, and many other diseases, even wrinkles and looking old. 
While we cannot stop all free radical production in our body, we can minimize our exposure and neutralize their effects by eating more antioxidants that stop free radicals from causing extensive damage. Antioxidants, found in a wide variety of fruits, especially deep colored fruits like blueberries, and vegetables (especially green leafy veggies), and some other foods, like apple cider vinegar, helps to reduce free radicals in the body.

2. Improves Satiety

Satiety refers to the feeling of being full and satisfied after a meal. Apple cider vinegar can help in this respect too, when taken before or with food.
When participants in a 2005 study were given varying levels of ACV along with carbohydrates in the form of white bread, the participants who had the most apple cider vinegar reported they felt fuller and were more satisfied after their meal than the others.
In addition, blood tests showed that the higher the levels of vinegar taken, the lower the blood glucose and insulin levels of participants.
Another study that same year found that adding apple cider vinegar (or peanuts!) to a meal helped to balance blood sugar levels, and improve feelings of fullness so much that the participants ate between 200 and 275 fewer calories during the rest of the day!
Increasing feelings of fullness without increasing caloric intake – as is the case with taking apple cider vinegar – is one of the most effective methods for losing weight over time.

3. Reduces Body Fat

Quite a bit of research shows that simply adding apple cider vinegar to the diet can reduce fat accumulation.
In animal studies, mice and rats were found to experience a reduction in cholesterol levels, triglycerides, blood pressure, body fat accumulation and liver fat when administered vinegar.
However, a human study confirms that it’s not just animals that experience these benefits. When obese individuals were given daily doses of vinegar over a 12 week period, they enjoyed reduced belly fat, waist circumference, lower blood triglycerides and weight loss.
Those who drank one tablespoon daily lost 2.6 pounds on average; while those who consumed two tablespoons each day shed an average of 3.7 pounds.
Although it’s a modest weight loss given the time period, it was achieved with no other lifestyle or dietary changes!

4. It’s Probiotic for Gut Health

The human gut is a complex system which exerts a great deal of control over the health of the whole body. In fact, it houses around 80% of our immune system.
Improving the health of the gut has been found to reduce low level inflammation and may even help prevent obesity.
Rats given lactic acid bacteria while in utero put on significantly less weight than other rats eating the same high-calorie diet. They also had lower levels of minor inflammation, which has been associated with obesity. Similarly, in human babies gut bacteria has been shown to impact weight. Several other studies have found that obese adults have different levels of gut bacteria to slim people.
The good news is that you can still address imbalances in the gut as an adult.
One study, published in the Journal of Clinical Investigation, suggests that it may be possible to use probiotic bacteria to treat obesity and other chronic diseases, while other research found that obese people were able to reduce their abdominal fat by nearly 5%, and their subcutaneous fat by over 3%, just by drinking a probiotic-rich beverage for 12 weeks.
You know what’s a great probiotic-rich drink? That’s right … apple cider vinegar!
Consume it regularly to enjoy better gut health and all the benefits that brings – from weight loss and better digestion to improved cognitive function and mood. Here are 13 more ways to improve your gut health naturally.




Some patients prefer these type of pills:





References:
1. The Journal of Membrane Biology

Volume 247, Issue 8pp 667–673



 2016 Jun;65(3):208-13. doi: 10.1016/j.ancard.2016.04.004. Epub 2016 May 18.

[Anti-obesogenic effect of apple cider vinegar in rats subjected to a high fat diet].

[Article in French]

Abstract

AIM OF THE STUDY:

The search of new anti-obesogenic treatments based on medicinal plants without or with minimal side effects is a challenge. In this context, the present study was conducted to evaluate the anti-obesogenic effect of apple cider vinegar (ACV) in Wistar rats subjected to a high fat diet.

MATERIALS AND METHODS:

Eighteen male Wistar rats (140±5g) were divided into 3 three equal groups. A witness group submitted to standard laboratory diet and two groups subjected to a high fat diet (cafeteria diet); one receives a daily gavage of apple cider vinegar (7mL/kg/d) for 30 days. Throughout the experiment monitoring the nutritional assessment, anthropometric and biochemical parameters is achieved.

RESULTS:

In the RCV vs RC group, we observed a highly significant decrease (P<0.001) in body weight and food intake. On the other hand, the VCP decreases very significantly different anthropometric parameters: BMI (P<0.01), chest circumference and abdominal circumference (P<0.001), decreases serum glucose levels (26.83%) and improves the serum lipid profile by reducing plasma levels of total cholesterol (34.29%), TG (51.06%), LDL-c (59.15%), VLDL (50%) and the total lipid (45.15%), and increasing HDL-c (39.39%), thus offering protection against oatherogenic risk (61.62%).

CONCLUSION:

This preliminary study indicates that the metabolic disorders caused by high fat diet (cafeteria) are thwarted by taking applecider vinegar which proves to have a satiating effect, antihyperlipidemic and hypoglycemic effects, and seems prevent the atherogenic risk.

 2014 Aug;247(8):667-73. doi: 10.1007/s00232-014-9685-5. Epub 2014 Jun 4.

Apple cider vinegar modulates serum lipid profile, erythrocyte, kidney, and liver membrane oxidative stress in ovariectomized mice fed high cholesterol.

Abstract

The purpose of this study was to investigate the potentially beneficial effects of apple cider vinegar (ACV) supplementation on serum triglycerides, total cholesterol, liver and kidney membrane lipid peroxidation, and antioxidant levels in ovariectomized (OVX) mice fed high cholesterol. Four groups of ten female mice were treated as follows: Group I received no treatment and was used as control. Group II was OVX mice. Group III received ACV intragastrically (0.6% of feed), and group IV was OVX and was treated with ACV as described for group III. The treatment was continued for 28 days, during which the mice were fed a high-cholesterol diet. The lipid peroxidation levels in erythrocyte, liver and kidney, triglycerides, total, and VLDL cholesterol levels in serum were higher in the OVX group than in groups III and IV. The levels of vitamin E in liver, the kidney and erythrocyte glutathione peroxidase (GSH-Px), and erythrocyte-reduced glutathione (GSH) were decreased in group II. The GSH-Px, vitamin C, E, and β-carotene, and the erythrocyte GSH and GSH-Px values were higher in kidney of groups III and IV, but in liver the vitamin E and β-carotene concentrations were decreased. In conclusion, ACV induced a protective effect against erythrocyte, kidney, and liver oxidative injury, and lowered the serum lipid levels in mice fed high cholesterol, suggesting that it possesses oxidative stress scavenging effects, inhibits lipid peroxidation, and increases the levels of antioxidant enzymes and vitamin.
 2016 Mar;54(1):113-119.

Comparison of Cultivable Acetic Acid Bacterial Microbiota in Organic and Conventional AppleCider Vinegar.

Abstract

Organic apple cider vinegar is produced from apples that go through very restricted treatment in orchard. During the first stage of the process, the sugars from apples are fermented by yeasts to cider. The produced ethanol is used as a substrate by acetic acid bacteria in a second separated bioprocess. In both, the organic and conventional apple cider vinegars the ethanol oxidation to acetic acid is initiated by native microbiota that survived alcohol fermentation. We compared the cultivable acetic acid bacterial microbiota in the production of organic and conventional apple cider vinegars from a smoothly running oxidation cycle of a submerged industrial process. In this way we isolated and characterized 96 bacteria from organic and 72 bacteria from conventional apple cider vinegar. Using the restriction analysis of the PCR-amplified 16S-23S rRNA gene ITS regions, we identified four different HaeIII and five different HpaII restriction profiles for bacterial isolates from organic apple cider vinegar. Each type of restriction profile was further analyzed by sequence analysis of the 16S-23S rRNA gene ITS regions, resulting in identification of the following species: Acetobacter pasteurianus (71.90%), Acetobacter ghanensis (12.50%), Komagataeibacter oboediens (9.35%) and Komagataeibacter saccharivorans (6.25%). Using the same analytical approach in conventional apple cider vinegar, we identified only two different HaeIII and two different HpaII restriction profiles of the 16S‒23S rRNA gene ITS regions, which belong to the species Acetobacter pasteurianus (66.70%) and Komagataeibacter oboediens (33.30%). Yeasts that are able to resist 30 g/L of acetic acid were isolated from the acetic acid production phase and further identified by sequence analysis of the ITS1-5.8S rDNA‒ITS2 region as Candida ethanolicaPichia membranifaciens and Saccharomycodes ludwigii. This study has shown for the first time that the bacterial microbiota for the industrial production of organic apple cider vinegar is clearly more heterogeneous than the bacterial microbiota for the industrial production of conventional apple cider vinegar. Further chemical analysis should reveal if a difference in microbiota composition influences the quality of different types of apple cider vinegar.


 2017 Jan;60:65-71. doi: 10.1016/j.fsi.2016.11.030. Epub 2016 Nov 10.

Dietary effect of apple cider vinegar and propionic acid on immune related transcriptional responses and growth performance in white shrimp, Litopenaeus vannamei.

Abstract

This experiment was conducted to study the effect of various levels of ACV® and Propionic acid (PA) on expression of immune related genes and growth performance in white shrimp (Litopenaeus vannamei). Three hundred and seventy-five shrimps with an average initial weight of 10.2 ± 0.04 g were collected and acclimatized for two weeks. Five experimental diets including control diet, 0.5% PA diet and 1%, 2% and 4% ACV® diets were applied to feed the shrimps. They were fed 4 times a day with 2.5% of body weight. After 60 days of culture, shrimps fed with ACV® and PA diets showed no significant difference in growth performance. Expression of prophenoloxidase (proPo), lysozyme (Lys), penaeidin-3a (Pen-3a) and Crustin (Cru) genes were determined from hepatopancreas, using the real-time PCR after 15, 30 and 60 days. Expression of Lys and proPo genes was significantly up regulated in shrimps fed with ACV® and PA diets compared to the control group after 30 and 60 days of treatment. After 15 days, Pen-3a gene expression was significantly higher in PA group compared to the control group. Also, shrimps fed with 1% and 4% ACV® and PA diets showed significantly increased expression of Pen-3a after 30 days. In contrast, expression of Cru was significantly down regulated in response to ACV® diets, but, Cru expression in treated shrimps with PA diet was greater than the control group after 30 and 60 days. Overall, the results provided evidence that ACV® could be used as a natural immunostimulant for shrimps in order to adjust and enhance expression of the immune related gene



 2009 Dec;29(12):846-9. doi: 10.1016/j.nutres.2009.10.021.

Vinegar lacks antiglycemic action on enteral carbohydrate absorption in human subjects.

Abstract

The antiglycemic effects of vinegar have been known for more than a century and have been demonstrated in animal as well as human studies. Although the exact mechanism of vinegar action is not known, several possibilities have been proposed including suppression of disaccharidase activity, delayed gastric emptying, enhanced glucose uptake in the periphery and conversion to glycogen, and increased satiety. We hypothesized that by suppressing endogenous insulin secretion, we could estimate the glucose absorption rate from an oral carbohydrate load and determine the effects of vinegar ingestion on this rate. To do so, 5 subjects had 4 studies at 1-week intervals, randomly receiving placebo twice (60 mL water) and vinegar twice (20 mL apple cider vinegar, 40 mL water), followed 2 minutes later by a meal of mashed potatoes (0.75 g carbohydrate per kilogram body weight) that was consumed over 20 minutes. At the beginning of the meal, an oral octreotide/insulin suppression test (25-microg bolus octreotide; 180 minute infusion 5 mU/m(2) body surface area per minute regular human insulin, and 0.5 microg/min octreotide) was begun. Blood samples for insulin and glucose were drawn at 20-minute intervals. The oral octreotide/insulin suppression test suppressed endogenous insulin secretion for the first 100 minutes of the study. During this time, the rate of rise of glucose was modestly but significantly (P = .01) greater after vinegar ingestion compared to placebo, suggesting that vinegar does not act to decrease glycemia by interference with enteral carbohydrate absorption.

 2015 Jun;24(4):296-302. doi: 10.1111/jopr.12207. Epub 2014 Sep 14.

Antifungal Activity of Apple Cider Vinegar on Candida Species Involved in Denture Stomatitis.

Abstract

PURPOSE:

To evaluate the in vitro antifungal activity of apple cider vinegar on Candida spp. involved in denture stomatitis.

MATERIAL AND METHODS:

The microdilution technique was used to determine the minimum inhibitory concentration (MIC) and minimum fungicidal concentration (MFC) of apple cider vinegar containing 4% maleic acid, and nystatin (control). Further tests of microbial kinetics and inhibition of adherence to acrylic resin were performed testing different concentrations (MIC, MICx2, MICx4) of the products at time intervals of 0, 30, 60, 120 and 180 minutes. A roughness meter was used to measure the changes in surface roughness; color change of the acrylic resin specimens exposed to the test products in different concentrations and time intervals were also evaluated.

RESULTS:

Apple cider vinegar (4%) showed MIC of 2500 μg/ml and MFC of 2500, 5000, and 10,000 μg/ml depending on the strain tested. Nystatin showed MIC of 3.125 μg/ml and strain-dependent MFC values ranging from 3.125 to 12.5 μg/ml. The microbial kinetic assay showed a statistical difference between apple cider vinegar and nystatin (p < 0.0001). After 30 minutes of exposure, apple cider vinegar showed fungicidal effect at MICx4, whereas nystatin maintained its fungistatic effect. Apple cider vinegar showed greater inhibition of adherence (p < 0.001) compared to control. Apple cider vinegar did not significantly alter the surface roughness of the acrylic resin specimens compared to nystatin (p > 0.05), and both had no influence on their color.

CONCLUSION:

Apple cider vinegar showed antifungal properties against Candida spp., thus representing a possible therapeutic alternative for patients with denture stomatitis.

DO NOT USE Apple Cider Vinegar to remove moles or freckles:

 2015 Jun;8(6):50.

Chemical Burn from Vinegar Following an Internet-based Protocol for Self-removal of Nevi.

Abstract

“Natural home remedies” for nevi removal found on the Internet can be ineffective, or worse, dangerous. Children and teens, in particular, may be more likely to attempt self-treatment in order to avoid discussing their concerns with their parents. Here, the authors report a case of an adolescent who presented with a chemical burn after following an Internet-based protocol for nevi removal using apple cider vinegar.

These are all the papers on Apple Cider Vinegar on Pubmed as of May 2017

Search results

Items: 53

1.
Frewin AJ, Renkema J, Fraser H, Hallett RH.
J Econ Entomol. 2017 Apr 21. doi: 10.1093/jee/tox081. [Epub ahead of print]
PMID:

 

28444365
2.
González Flores M, Rodríguez ME, Oteiza JM, Barbagelata RJ, Lopes CA.
Int J Food Microbiol. 2017 May 16;249:9-17. doi: 10.1016/j.ijfoodmicro.2017.02.018. Epub 2017 Mar 1.
PMID:

 

28271856
3.
Štornik A, Skok B, Trček J.
Food Technol Biotechnol. 2016 Mar;54(1):113-119.
PMID:

 

27904401

 

Free PMC Article

4.
Pourmozaffar S, Hajimoradloo A, Miandare HK.
Fish Shellfish Immunol. 2017 Jan;60:65-71. doi: 10.1016/j.fsi.2016.11.030. Epub 2016 Nov 10.
PMID:

 

27840173

 

Free Article

5.
Akasaka N, Higashikubo H, Ishii Y, Sakoda H, Fujiwara S.
J Biosci Bioeng. 2017 Jan;123(1):78-83. doi: 10.1016/j.jbiosc.2016.06.014. Epub 2016 Aug 31.
PMID:

 

27591976
6.
Bouderbala H, Kaddouri H, Kheroua O, Saidi D.
Ann Cardiol Angeiol (Paris). 2016 Jun;65(3):208-13. doi: 10.1016/j.ancard.2016.04.004. Epub 2016 May 18. French.
PMID:

 

27209492
7.
Trček J, Mahnič A, Rupnik M.
Int J Food Microbiol. 2016 Apr 16;223:57-62. doi: 10.1016/j.ijfoodmicro.2016.02.007. Epub 2016 Feb 10.
PMID:

 

26897250
8.
Lin CC, Prokop-Prigge KA, Preti G, Potter CJ.
Elife. 2015 Sep 30;4. pii: e08688. doi: 10.7554/eLife.08688.
PMID:

 

26422512

 

Free PMC Article

9.
Burrack HJ, Asplen M, Bahder L, Collins J, Drummond FA, Guédot C, Isaacs R, Johnson D, Blanton A, Lee JC, Loeb G, Rodriguez-Saona C, van Timmeren S, Walsh D, McPhie DR.
Environ Entomol. 2015 Jun;44(3):704-12. doi: 10.1093/ee/nvv022. Epub 2015 Mar 21.
PMID:

 

26313977
10.
Feldstein S, Afshar M, Krakowski AC.
J Clin Aesthet Dermatol. 2015 Jun;8(6):50.
PMID:

 

26155328

 

Free PMC Article

11.
Joshi NK, Biddinger DJ, Demchak K, Deppen A.
J Insect Sci. 2014 Jan 1;14:259. doi: 10.1093/jisesa/ieu121. Print 2014.
PMID:

 

25434039

 

Free PMC Article

12.
Mota AC, de Castro RD, de Araújo Oliveira J, de Oliveira Lima E.
J Prosthodont. 2015 Jun;24(4):296-302. doi: 10.1111/jopr.12207. Epub 2014 Sep 14.
PMID:

 

25219289
13.
Iglesias LE, Nyoike TW, Liburd OE.
J Econ Entomol. 2014 Aug;107(4):1508-18.
PMID:

 

25195443
14.
Brown R, Sam CH, Green T, Wood S.
J Diet Suppl. 2015 Jun;12(2):138-45. doi: 10.3109/19390211.2014.950783. Epub 2014 Aug 21.
PMID:

 

25144853
15.
Nazıroğlu M, Güler M, Özgül C, Saydam G, Küçükayaz M, Sözbir E.
J Membr Biol. 2014 Aug;247(8):667-73. doi: 10.1007/s00232-014-9685-5. Epub 2014 Jun 4.
PMID:

 

24894721
16.
Hamby KA, Bolda MP, Sheehan ME, Zalom FG.
Environ Entomol. 2014 Aug;43(4):1008-18. doi: 10.1603/EN13245. Epub 2014 May 26.
PMID:

 

24865227
17.
Kleiber JR, Unelius CR, Lee JC, Suckling DM, Qian MC, Bruck DJ.
Environ Entomol. 2014 Apr;43(2):439-47. doi: 10.1603/EN13224.
PMID:

 

24763098
18.
Belien T, Thys T, Fassotte C, Walrant C, Tomme M, Bolen M, Bylemans D.
Commun Agric Appl Biol Sci. 2014;79(2):169-75.
PMID:

 

26084094
19.
Cha DH, Hesler SP, Cowles RS, Vogt H, Loeb GM, Landolt PJ.
Environ Entomol. 2013 Oct;42(5):1052-60. doi: 10.1603/EN13154.
PMID:

 

24331615
20.
Lee JC, Shearer PW, Barrantes LD, Beers EH, Burrack HJ, Dalton DT, Dreves AJ, Gut LJ, Hamby KA, Haviland DR, Isaacs R, Nielsen AL, Richardson T, Rodriguez-Saona CR, Stanley CA, Walsh DB, Walton VM, Yee WL, Zalom FG, Bruck DJ.
Environ Entomol. 2013 Dec;42(6):1348-55. doi: 10.1603/EN13148. Epub 2013 Nov 18.
PMID:

 

24252375
21.
22.
Faucher CP, Hilker M, de Bruyne M.
PLoS One. 2013;8(2):e56361. doi: 10.1371/journal.pone.0056361. Epub 2013 Feb 15.
PMID:

 

23457557

 

Free PMC Article

23.
Lee H, Kim H, Cadwallader KR, Feng H, Martin SE.
Ultrason Sonochem. 2013 Jul;20(4):1131-8. doi: 10.1016/j.ultsonch.2013.01.003. Epub 2013 Jan 22.
PMID:

 

23395259
24.
Gambon DL, Brand HS, Veerman EC.
Ned Tijdschr Tandheelkd. 2012 Dec;119(12):589-91. Dutch.
PMID:

 

23373303
25.
Bunick CG, Lott JP, Warren CB, Galan A, Bolognia J, King BA.
J Am Acad Dermatol. 2012 Oct;67(4):e143-4. doi: 10.1016/j.jaad.2011.11.934. No abstract available.
PMID:

 

22980269
26.
Gaston A, Garry RF.
Virol J. 2012 Jan 17;9:21. doi: 10.1186/1743-422X-9-21.
PMID:

 

22251397

 

Free PMC Article

27.
Lu HJ, Breidt F Jr, Pérez-Díaz IM, Osborne JA.
J Food Prot. 2011 Jun;74(6):893-8. doi: 10.4315/0362-028X.JFP-10-404.
PMID:

 

21669064
28.
Budak NH, Kumbul Doguc D, Savas CM, Seydim AC, Kok Tas T, Ciris MI, Guzel-Seydim ZB.
J Agric Food Chem. 2011 Jun 22;59(12):6638-44. doi: 10.1021/jf104912h. Epub 2011 May 18.
PMID:

 

21561165
29.
Dornelles-Morgental R, Guerreiro-Tanomaru JM, de Faria-Júnior NB, Hungaro-Duarte MA, Kuga MC, Tanomaru-Filho M.
Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 2011 Sep;112(3):396-400. doi: 10.1016/j.tripleo.2011.02.004. Epub 2011 Apr 30.
PMID:

 

21531598
30.
Lee R.
Harv Heart Lett. 2009 Oct;20(2):8. No abstract available.
PMID:

 

20568348
31.
Salbe AD, Johnston CS, Buyukbese MA, Tsitouras PD, Harman SM.
Nutr Res. 2009 Dec;29(12):846-9. doi: 10.1016/j.nutres.2009.10.021.
PMID:

 

19963157
32.
Shishehbor F, Mansoori A, Sarkaki AR, Jalali MT, Latifi SM.
Pak J Biol Sci. 2008 Dec 1;11(23):2634-8.
PMID:

 

19630216
33.
Semmelhack JL, Wang JW.
Nature. 2009 May 14;459(7244):218-23. doi: 10.1038/nature07983. Epub 2009 Apr 26.
PMID:

 

19396157

 

Free PMC Article

34.
Garai-Ibabe G, Ibarburu I, Berregi I, Claisse O, Lonvaud-Funel A, Irastorza A, Dueñas MT.
Int J Food Microbiol. 2008 Feb 10;121(3):253-61. doi: 10.1016/j.ijfoodmicro.2007.11.004. Epub 2007 Nov 12.
PMID:

 

18180066
35.
Hlebowicz J, Darwiche G, Björgell O, Almér LO.
BMC Gastroenterol. 2007 Dec 20;7:46.
PMID:

 

18093343

 

Free PMC Article

36.
Breidt F Jr, Hayes J, McFeeters RF.
J Food Prot. 2007 Nov;70(11):2638-41.
PMID:

 

18044448
37.
del Campo G, Berregi I, Santos JI, Dueñas M, Irastorza A.
Bioresour Technol. 2008 May;99(8):2857-63. Epub 2007 Aug 13.
PMID:

 

17706419
38.
Xu K, Song J, Ren Y, Ma H, Huang J, Du X.
Se Pu. 2007 Jan;25(1):93-5. Chinese.
PMID:

 

17432584
39.
Mattila P, Hellström J, Törrönen R.
J Agric Food Chem. 2006 Sep 20;54(19):7193-9.
PMID:

 

16968082
40.
Faucher C, Forstreuter M, Hilker M, de Bruyne M.
J Exp Biol. 2006 Jul;209(Pt 14):2739-48.
PMID:

 

16809465

 

Free Article

41.
Bjornsdottir K, Breidt F Jr, McFeeters RF.
Appl Environ Microbiol. 2006 Jan;72(1):660-4.
PMID:

 

16391104

 

Free PMC Article

42.
Hill LL, Woodruff LH, Foote JC, Barreto-Alcoba M.
J Am Diet Assoc. 2005 Jul;105(7):1141-4.
PMID:

 

15983536
44.
45.
Vijayakumar C, Wolf-Hall CE.
J Food Prot. 2002 Oct;65(10):1646-50.
PMID:

 

12380754
46.
47.
Mayerhauser CM.
J Food Prot. 2001 Jun;64(6):783-7.
PMID:

 

11403126
48.
[No authors listed]
Posit Health News. 1998 Fall;(No 17):14-5.
PMID:

 

11366543
50.
Ralston SL.
Vet Clin North Am Equine Pract. 1990 Aug;6(2):339-54. Review.
PMID:

 

2202496
51.
Wolman PG.
J Am Diet Assoc. 1987 Sep;87(9):1211-4.
PMID:

 

3624710
52.
Krueger DA, Krueger HW.
J Assoc Off Anal Chem. 1985 May-Jun;68(3):449-52.
PMID:

 

4019367
53.
Banna AA, Kawar NS.
J Environ Sci Health B. 1982;17(5):505-14.
PMID:

 

7175099
Shopping Cart